I inspected the tiny creature closely. It must have died recently, its brown and white furred body still soft and supple. Was it an outdoor cat that killed it? It certainly could have been. Free roaming cats have left quite a number of birds and small mammals for me over the years. But there didn’t appear to be any signs of trauma; no puncture wound and no blood. Hmmm. I dismissed the thought that this animal was poisoned—surely my immediate neighbors know that rat bait is a wretched solution? I left the mouse where it was, figuring it could be food for some other critter. Unfortunately, this was a mistake.
The Big Lie
It turns out a neighbor did have a rodent bait box on his property. He explained that he had mice entering his house and insects that needed controlling which prompted him to hire a pest control company. When I mentioned the effects of poisoned rodents on our foxes and other wildlife, my neighbor, who equally enjoys our wildlife, insisted the bait used wasn’t poisonous—the exterminator told him so. Hmmm?! Evidently some pesticide companies tell customers only what they want to hear, no matter how big the lie.
Second-Generation Killers cause Secondary Victims
Today’s most widely used rat poisons are second-generation anticoagulants: highly toxic not only to the rodents that directly consume the bait but also to the non-target animals that eat the contaminated rodents.
Although the EPA has prohibited the sale of products containing brodifacoum, bromadiolone, difenacoum and difethialone directly to consumers, pest control companies and farmers are still allowed to use these second-gen anticoagulant rodenticides.
Like the first-generation of anticoagulants that preceded them, the “superwarfarins” prevent blood from clotting, induce internal bleeding and inflict a prolonged, painful death. However the newer poisons are many times more potent. “Second-generation anticoagulants,” the EPA explains, “are more likely than first-generation anticoagulants to be able to kill after a single night’s feeding. These compounds kill over a similar course of time but tend to remain in animal tissues longer than do first-generation ones.” This means that by the time the rodents die—five to seven days after consuming the bait—they can have up to 30-40 times the lethal dose in their bodies. Meanwhile, they stumble about and are easy prey for predators and pets.
Collateral poisonings of carnivores and omnivores alike are widespread wherever bait boxes are being deployed. The affected wildlife are more likely to get hit by moving vehicles, crash into structures or be killed by other animals. These non-target animals are also more susceptible to disease and vermin. In California, most coyotes, bobcats and cougars that die from mange—a skin disease caused by parasitic mites—also test positive for rodenticide exposure.
Some Fast Facts:
- Rodenticides are indiscriminate killers that attract and kill all kinds of animals, not just rats and mice.
- Second-generation anticoagulants are persistent and bioaccumulative; they remain in the victim’s bloodstream and accumulate in the liver.
- Non-target predators like foxes, coyotes, owls and hawks, and scavengers such as vultures, raccoons and opossums, suffer lethal and sub-lethal poisoning when they feed on poisoned rodents.
- Where second-generation anticoagulants are used, entire food webs are contaminated.
- By killing off the predators that would otherwise control rodents, anticoagulants actually generate rodent infestations.
- In one Canadian study of dead raptors, nearly 100 percent of owls had at least one anticoagulant rodenticide in their livers.
The Centers For Disease Control receive about 15,000 calls per year from parents whose children have eaten rodenticides.
- Cats and dogs are also harmed from eating poisoned rodents.